Disease Info Card

Atypical Absence Seizure

Information about Atypical Absence Seizure: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Atypical Absence Seizure

Most recent studies have shown that Atypical Absence Seizure shares some biological mechanisms with absence-epilepsy, atonic-seizures, complex-partial-seizures, convulsive-seizures, encephalopathies, epilepsies-myoclonic, epilepsies-partial, epilepsy, epilepsy-generalized, generalized-seizures, infantile-spasms, intractable-epilepsy, lennox-gastaut-syndrome, partial-seizure, spasm, status-epilepticus, tonic-clonic-epilepsy, tonic-convulsion.

Among the many pathways, these few ones have gauged particular interests from scientists studying Atypical Absence Seizure, and have been seen in publications frequently: Aging, Brain Development, Cell Proliferation, Cognition, Glucose Transport, Neurogenesis, Pathogenesis, Proestrus, Reflex, Synaptic Transmission, Transport

Quite a number of genes have been found to play important roles in Atypical Absence Seizure, such as BCL9, ERG, EXOSC7, EXT1, FGD1, FOS, FSCN1, HTN3, IL2RA, MCAT, MPP1, NUPR1, P4HB, POLG2, PSMD12, S100A8, SCN1A, SPINT2, TNFRSF1A, TPPP3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Atypical Absence Seizure Related Genes

click to see detail information for each gene

BCL9 ERG EXOSC7
EXT1 FGD1 FOS
FSCN1 HTN3 IL2RA
MCAT MPP1 NUPR1
P4HB POLG2 PSMD12
S100A8 SCN1A SPINT2
TNFRSF1A TPPP3

Pathways Related to Atypical Absence Seizure

This information is being compiled and will come in a future update

Aging Brain Development Cell Proliferation
Cognition Glucose Transport Neurogenesis
Pathogenesis Proestrus Reflex
Synaptic Transmission Transport