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- Table of Contents
Facts about Transcription factor RelB.
Different dimer combinations act as transcriptional activators or repressors, respectively. NF-kappa-B is controlled by various mechanisms of post-translational modification and subcellular compartmentalization in addition to by interactions with other cofactors or corepressors.
| Human | |
|---|---|
| Gene Name: | RELB |
| Uniprot: | Q01201 |
| Entrez: | 5971 |

| Belongs to: |
|---|
| No superfamily |

IREL; I-Rel; nuclear factor of kappalight polypeptide gene enhancer in B-cells 3; RelB; v-rel reticuloendotheliosis viral oncogene homolog B
Mass (kDA):
62.134 kDA

| Human | |
|---|---|
| Location: | 19q13.32 |
| Sequence: | 19; NC_000019.10 (45001449..45038194) |
Nucleus. Cytoplasm, cytoskeleton, microtubule organizing center, centrosome. Colocalizes with NEK6 in the centrosome.





PMID: 1577270 by Ruben S.M., et al. I-Rel: a novel rel-related protein that inhibits NF-kappa B transcriptional activity.
PMID: 1732739 by Ryseck R.P., et al. RelB, a new Rel family transcription activator that can interact with p50-NF-kappa B.