Zinc finger protein 592 Antibodies

AND ZNF592 ELISA kits, Zinc finger protein 592 Proteins

Many biological assays use antibodies to detect Zinc finger protein 592, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Zinc finger protein 592 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Zinc finger protein 592 antibodies...

We validate the specificity of these antibodies to Zinc finger protein 592 by testing them on tissues known to express ZNF592 positively and negatively. Browse below to find the ZNF592 antibody that suites your experiment. We have 1 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

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ZNF592 Infographic by Boster Bio

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Anti-Zinc finger protein 592 ZNF592 Antibody

1 Q&As

Immunohistochemistry analysis of paraffin-embedded human colon carcinoma tissue, using ZNF592 Antibody

Western blot analysis of lysates from K562 cells, using ZNF592 Antibody

Human, Mouse

ELISA, IF, IHC, ICC, WB

$335
Cat # A12580
100ul

ZNF592 Overview

Facts about Zinc finger protein 592.

ZNF592 3D structure. Source: alphafold

Zinc finger protein 592 (ZNF592)

May be involved in transcriptional regulation. .

Gene Information

Human
Gene Name:	ZNF592
Uniprot:	Q92610
Entrez:	9640

Family

Belongs to:

krueppel C2H2-type zinc-finger protein family

Alternative Names

CAMOS; KIAA0211MGC138437; MGC138439; SCAR5; spinocerebellar ataxia, autosomal recessive 5; zinc finger protein 592

Molecular Weight

Mass (kDA):
137.528 kDA

Genomic Context


Human

Location:	15q25.3
Sequence:	15; NC_000015.10 (84748556..84806445)

Tissue Specificity

Widely expressed, with highest levels in skeletal muscle. Expressed throughout the central nervous system, including in the cerebellum and cerebellar vermis, with higher expression in the substantia nigra. Widely expressed in fetal tissues.

Subcellular Localizations

Nucleus.

Diseases

Optic Atrophy
Lymphomatoid Papulosis
Ataxia
Cerebellar Ataxia
Atrophy
Atherosclerosis
Autoimmune Diseases
Coronary Artery Disease
Arthritis
Heart Diseases
Autoimmune Reaction

Skin Abnormalities
Spinocerebellar Degeneration
Cardiac Arrest
Rheumatoid Arthritis
Sudden Cardiac Death
Sudden Cardiac Arrest

Interacting Proteins

TCAF1

Pathways

Immune Response

PTMs

Isopeptide bond

Phosphoprotein

Ubl conjugation

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/ZNF592

References

PMID: 20531441 by Nicolas E., et al. CAMOS, a nonprogressive, autosomal recessive, congenital cerebellar ataxia, is caused by a mutant zinc-finger protein, ZNF592.