Disease Info Card

Persistent Fetal Circulation Syndrome

Information about Persistent Fetal Circulation Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Persistent Fetal Circulation Syndrome

Most recent studies have shown that Persistent Fetal Circulation Syndrome shares some biological mechanisms with anoxia, congenital-diaphragmatic-hernia, congenital-heart-defects, diaphragmatic-hernia, fetal-diseases, fetal-growth-retardation, heart-diseases, hemorrhage, hernia, hypertensive-disease, hypoxia, lung-diseases, meconium-aspiration-syndrome, pathologic-vasoconstriction, pre-eclampsia, pregnancy-complications, pulmonary-hypertension, respiratory-distress, respiratory-distress-syndrome-newborn, respiratory-failure.

Among the many pathways, these few ones have gauged particular interests from scientists studying Persistent Fetal Circulation Syndrome, and have been seen in publications frequently: Amino Acid Transport, Angiogenesis, Cell Death, Cell Proliferation, Coagulation, Excretion, Hormone Secretion, Immune Response, Inflammatory Response, Localization, Lung Development, Lung Growth, Parturition, Pathogenesis, Platelet Aggregation, Secretion, Transport, Transposition, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Persistent Fetal Circulation Syndrome, such as AGT, ALB, AVP, CALR, CRH, EDN1, FUT2, IGF1, IL6, INS, NOS2, NOS3, POMC, SQLE, SSB, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.