Sideroflexin-1 Antibodies

AND Sfxn1 ELISA kits, Sideroflexin-1 Proteins

Many biological assays use antibodies to detect Sideroflexin-1, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Sideroflexin-1 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Sideroflexin-1 antibodies...

We validate the specificity of these antibodies to Sideroflexin-1 by testing them on tissues known to express Sfxn1 positively and negatively. Browse below to find the Sfxn1 antibody that suites your experiment. We have 2 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

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Sfxn1 Infographic by Boster Bio

All Sfxn1 Products

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SFXN1 (NM_022754) Human Over-expression Lysate

Human

$628
Cat # LS094081
20 µg, 100 µg

Anti-SFXN1 Antibody Picoband®

Western blot analysis of SFXN1 using anti-SFXN1 antibody (A11524-1)

IHC analysis of SFXN1 using anti-SFXN1 antibody (A11524-1)

Human, Mouse, Rat

Flow Cytometry, IP, IF, IHC, ICC, WB

$370
Cat # A11524-1
100 μg/vial

Sfxn1 Overview

Facts about Sideroflexin-1.

Sideroflexin-1 (Sfxn1)

Might be involved in the transport of a part needed for iron utilization into or from the mitochondria. .

Gene Information

Mouse
Gene Name:	Sfxn1
Uniprot:	Q99JR1
Entrez:	14057

Family

Belongs to:

sideroflexin family

Alternative Names

FLJ12876; sideroflexin 1; sideroflexin-1; TCC; Tricarboxylate carrier protein

Molecular Weight

Mass (kDA):
35.649 kDA

Genomic Context


Mouse

Location:	13 B1\|13 28.4 cM
Sequence:	13;

Tissue Specificity

Widely expressed, with highest expression in kidney and liver.

Diseases

Anemia
Sideroblastic Anemia
Friedreich Ataxia
Anemia, Neonatal
Insulinoma
Nerve Degeneration
Iron Overload
Ataxia
Hereditary Hemochromatosis

Iron Metabolism Disorders
Nervousness
Ceruloplasmin Deficiency
Hemochromatosis

Pathways

Transport
Regeneration
Insulin Secretion
Secretion

PTMs

Acetylation

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/Sfxn1

References

PMID: 11274051 by Fleming M.D., et al. A mutation in a mitochondrial transmembrane protein is responsible for the pleiotropic hematological and skeletal phenotype of flexed- tail (f/f) mice.