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- Table of Contents
Ahrr Antibodies
AND Ahrr ELISA kits, Ahrr Proteins
Many biological assays use antibodies to detect Ahrr, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Ahrr in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Ahrr antibodies...
Designing a Western blot for Ahrr? The guide has the expected band size, antibodies backed by real blot images, the controls to run alongside, and protocols taken from published papers.
Open the AHRR Western Blot GuideAhrr Infographic by Boster Bio
All Ahrr Products
Product
Figures
Specificity
Applications
Price
Human
ELISA, Flow Cytometry, IF, IHC, ICC, WB
$370
Cat # A01515-3
100 µg/vial
Cat # A01515-3
100 µg/vial
Ahrr Overview
Facts about Aryl hydrocarbon receptor repressor.
Aryl hydrocarbon receptor repressor (Ahrr)
Mediates dioxin toxicity and is involved in regulation of cell growth and differentiation. Represses the transcription activity of AHR by competing with this transcription factor for heterodimer formation with the ARNT and then binding to the xenobiotic response element (XRE) sequence within the promoter regulatory region of variety of genes.
Represses CYP1A1 by binding the XRE sequence and recruiting ANKRA2, HDAC4 and/or HDAC5. Autoregulates its expression by associating with its XRE site.
Gene Information
| Mouse | |
|---|---|
| Gene Name: | Ahrr |
| Uniprot: | Q3U1U7 |
| Entrez: | 11624 |
Family
| Belongs to: |
|---|
| No superfamily |
Alternative Names
Aryl hydrocarbon receptor repressor
Molecular Weight
Mass (kDA):
77.726 kDA
Genomic Context
| Mouse | |
|---|---|
| Location: | 13|13 C1 |
| Sequence: | 13; |
PTMs
- Isopeptide bond
For details, visit:
bosterbio.com/bosterbio-gene-info-cards/Ahrr
bosterbio.com/bosterbio-gene-info-cards/Ahrr
References
PMID: 9887096 by Mimura J., et al. Identification of a novel mechanism of regulation of Ah (dioxin) receptor function.
PMID: 12520768 by Huang P., et al. Constitutive and TCDD-induced expression of Ah receptor-responsive genes in the pituitary.