Zbtb49 Antibodies

AND Zbtb49 ELISA kits, Zbtb49 Proteins

Many biological assays use antibodies to detect Zbtb49, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Zbtb49 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Zbtb49 antibodies...

We validate the specificity of these antibodies to Zbtb49 by testing them on tissues known to express Zbtb49 positively and negatively. Browse below to find the Zbtb49 antibody that suites your experiment. We have 1 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

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Zbtb49 Infographic by Boster Bio

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ZNF509 (ZBTB49) (NM_145291) Human Over-expression Lysate

Human

$960
Cat # LS166793
20 µg, 100 µg

Zbtb49 Overview

Facts about Zinc finger and BTB domain-containing protein 49.

Zinc finger and BTB domain-containing protein 49 (Zbtb49)

Transcription element. Inhibits cell proliferation by activating either CDKN1A/p21 transcription or RB1 transcription.

Gene Information

Mouse
Gene Name:	Zbtb49
Uniprot:	Q8BXX2
Entrez:	75079

Family

Belongs to:

krueppel C2H2-type zinc-finger protein family

Alternative Names

FLJ38559; MGC126279; zinc finger and BTB domain containing 49; zinc finger and BTB domain-containing protein 49; Zinc finger protein 509MGC126280; ZNF509

Molecular Weight

Mass (kDA):
83.091 kDA

Genomic Context


Mouse

Location:	5\|5 B3
Sequence:	5;

Tissue Specificity

Widely expressed, with highest levels in white adipose tissue and kidney, intermediate levels in brain, liver and heart, and lowest levels in spleen, brown adipose tissue and muscle.

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/Zbtb49

References

PMID: 25245946 by Jeon B.N., et al. Two ZNF509 (ZBTB49) isoforms induce cell-cycle arrest by activating transcription of p21/CDKN1A and RB upon exposure to genotoxic stress.