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Product Info Summary
|Reactive Species:||Human, Mouse, Rat|
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Anti-Nephrocystin-5 (R467) IQCB1 Antibody
Boster Bio Anti-Nephrocystin-5 (R467) IQCB1 Antibody catalog # A07108-1. Tested in IHC, WB applications. This antibody reacts with Human, Mouse, Rat.
Storage & Handling
Store at -20°C for one year. For short term storage and frequent use, store at 4°C for up to one month. Avoid repeated freeze-thaw cycles.
Cite This Product
Anti-Nephrocystin-5 (R467) IQCB1 Antibody (Boster Biological Technology, Pleasanton CA, USA, Catalog # A07108-1)
Rabbit IgG, 1mg/ml in PBS with 0.02% sodium azide, 50% glycerol, pH7.2
A07108-1 is reactive to IQCB1 in Human, Mouse, Rat
A07108-1 is guaranteed for IHC, WB Boster Guarantee
Boster validates all antibodies on WB, IHC, ICC, Immunofluorescence and ELISA with known positive and negative samples to ensure specificity and high affinity.
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Assay Dilutions Recommendation
The recommendations below provide a starting point for assay optimization. Actual working concentration varies and should be decided by the user.
WB: 1:500~1:1000 IHC: 1:50~1:200
Validation Images & Assay Conditions
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Gene/Protein Information For IQCB1 (Source: Uniprot.Org, NCBI)
IQ calmodulin-binding motif-containing protein 1
IQ calmodulin-binding motif containing 1; IQ motif containing B1; KIAA0036nephrocystin 5; Nephrocystin-5; NPHP5IQ calmodulin-binding motif-containing protein 1; p53 and DNA damage-regulated IQ motif protein; PIQ; SLSN5 IQCB1 NPHP5, PIQ, SLSN5 IQ motif containing B1 IQ calmodulin-binding motif-containing protein 1|nephrocystin 5|p53 and DNA damage-regulated IQ motif protein*if product is indicated to react with multiple species, protein info is based on the gene entry specified above in "species".
For more info on IQCB1, check out the IQCB1 Infographic
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In this infographic you will see the following information for IQCB1: database IDs, super-family, protein function, synonyms, molecular weight, chromosomal locations, tissues of expression, subcellular locations, post translational modifications, and related diseases, research areas & pathways. If you want to see more information included, or would like to contribute to it and be acknowledged, please contact us [email protected]
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